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https://lib.hpu.edu.vn/handle/123456789/23549
Full metadata record
DC Field | Value | Language |
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dc.contributor.author | Gutierrez-Mazariegos, Juliana | en_US |
dc.date.accessioned | 2016-10-11T05:37:12Z | |
dc.date.available | 2016-10-11T05:37:12Z | |
dc.date.issued | 2016 | en_US |
dc.identifier.other | HPU4160660 | en_US |
dc.identifier.uri | https://lib.hpu.edu.vn/handle/123456789/23549 | en_US |
dc.description.abstract | Whole genome duplications (WGDs) have been classically associated with the origin of evolutionary novelties and the so-called duplication–degeneration–complementation model describes the possible fates of genes after duplication. However, how sequence divergence effectively allows functional changes between gene duplicates is still unclear. In the vertebrate lineage, two rounds of WGDs took place, giving rise to paralogous gene copies observed for many gene families. For the retinoic acid receptors (RARs), for example, which are members of the nuclear hormone receptor (NR) superfamily, a unique ancestral gene has been duplicated | en_US |
dc.format.extent | 15 p. | en_US |
dc.format.mimetype | application/pdf | en_US |
dc.language.iso | en | en_US |
dc.subject | Biology | en_US |
dc.subject | Developmental biology | en_US |
dc.subject | Evolution | en_US |
dc.subject | Cyclostomes | en_US |
dc.subject | Emergence of evolutionary novelty | en_US |
dc.subject | Gene andwhole genome duplication | en_US |
dc.subject | Hagfish and lamprey | en_US |
dc.subject | Nuclear hormone receptor signalling | en_US |
dc.title | Evolutionary diversification of retinoic acid receptor ligand-binding pocket structure bymolecular tinkering | en_US |
dc.type | Article | en_US |
dc.size | 1.27MB | en_US |
dc.department | Education | en_US |
Appears in Collections: | Education |
Files in This Item:
File | Description | Size | Format | |
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0543_Evolutionary.pdf Restricted Access | 1.3 MB | Adobe PDF | View/Open Request a copy |
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