Please use this identifier to cite or link to this item: https://lib.hpu.edu.vn/handle/123456789/23549
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dc.contributor.authorGutierrez-Mazariegos, Julianaen_US
dc.date.accessioned2016-10-11T05:37:12Z
dc.date.available2016-10-11T05:37:12Z
dc.date.issued2016en_US
dc.identifier.otherHPU4160660en_US
dc.identifier.urihttps://lib.hpu.edu.vn/handle/123456789/23549en_US
dc.description.abstractWhole genome duplications (WGDs) have been classically associated with the origin of evolutionary novelties and the so-called duplication–degeneration–complementation model describes the possible fates of genes after duplication. However, how sequence divergence effectively allows functional changes between gene duplicates is still unclear. In the vertebrate lineage, two rounds of WGDs took place, giving rise to paralogous gene copies observed for many gene families. For the retinoic acid receptors (RARs), for example, which are members of the nuclear hormone receptor (NR) superfamily, a unique ancestral gene has been duplicateden_US
dc.format.extent15 p.en_US
dc.format.mimetypeapplication/pdfen_US
dc.language.isoenen_US
dc.subjectBiologyen_US
dc.subjectDevelopmental biologyen_US
dc.subjectEvolutionen_US
dc.subjectCyclostomesen_US
dc.subjectEmergence of evolutionary noveltyen_US
dc.subjectGene andwhole genome duplicationen_US
dc.subjectHagfish and lampreyen_US
dc.subjectNuclear hormone receptor signallingen_US
dc.titleEvolutionary diversification of retinoic acid receptor ligand-binding pocket structure bymolecular tinkeringen_US
dc.typeArticleen_US
dc.size1.27MBen_US
dc.departmentEducationen_US
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